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抑制剂显著逆转,提示芍药苷可通过激活AMPK/Nrf2通                            的溃疡性结肠炎小鼠的改善作用[J]. 中国新药杂志,
          路来抑制氧化应激,从而改善小鼠UC。                                      2023,32(3):276-282.
              综上所述,芍药苷可修复小鼠的肠上皮细胞损伤,                              CHENG  N  N,ZHENG  C  X,DAI  X,et  al.  Protective
          改善肠上皮细胞间紧密连接,并上调相关蛋白、促抗氧                                effect of baicalein on mice with ulcerative colitis induced
          化分子的表达和释放,从而改善UC;其作用机制可能与                               by  dextran  sulfate  sodium  salt[J].  Chin  J  New  Drugs,
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          激活 AMPK/Nrf2 抗氧化通路有关。但芍药苷对 UC 的
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          中国药房  2025年第36卷第4期                                                 China Pharmacy  2025 Vol. 36  No. 4    · 433 ·
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