Page 145 - 2019年2月第30卷第4期
P. 145

diseases in clinical chemistry[J]. Clin Biochem,2014,47  and non-M. tuberculosis:a summary[J]. Biomed Res Int,
             (15):16-26.                                         2015. DOI:10.1155/2015/895453.
        [ 5 ]  秦昆明,王彬,陈林伟,等.代谢组学在中药现代研究的应                   [20]  GOUZY A,LARROUY-MAUMUS G,BOTTAI D,et al.
             用与展望[J].中国中药杂志,2014,39(16):3010-3017.               Mycobacterium tuberculosis exploits asparagine to assimi-
        [ 6 ]  纪勇,郭盛磊,杨玉焕.代谢组学方法研究进展[J].安徽农                      late nitrogen and resist acid stress during infection[J].
             业科学,2015,43(25):21-23.                              PLoS Pathog,2014. DOI:10.1371/journal.ppat.1003928.
        [ 7 ]  CHUANG YM,DUTTA NK,HUNG CF,et al. Stringent  [21]  周宁,杜祥博,杨莉.不同病程的结核病患者的血浆代谢
             response factors PPX1 and PPK2 play an important role  组学研究[J].波谱学杂志,2016,33(2);224-235.
             in Mycobacterium tuberculosis metabolism,biofilm for-  [22]  张宗德,李自慧,杜博平,等.结核杆菌体内诱导基因的筛
             mation,and sensitivity to isoniazid in vivo[J]. Antimicrob  选及初步分析[J].结核病与胸部肿瘤,2008(3):161-162.
             Agents Chemother,2016,60(11):6460-6470.        [23]  ANGELI A,DEL PRETE S,OSMAN SM,et al. Activa-
        [ 8 ]  冯鹭,孟繁超.海绵附生放线菌抗结核分枝杆菌活性物质                         tion studies with amines and amino acids of the betacar-
             的筛选及分离[J].山东大学学报(医学版),2016,54(5):                   bonic anhydrase encoded by the Rv3273 gene from the
             29-33.                                              pathogenic bacterium Mycobacterium tuberculosis[J]. J
        [ 9 ]  周宁,杜祥博,杨莉,等.不同病程的结核病患者的血浆代                        Enzyme Inhib Med Chem,2018,33(1):364-369.
             谢组学研究[J].波谱学杂志,2016,33(2):224-225.             [24]  NOY T,VERGNOLLE O,HARTMAN TE,et al. Central
        [10]  董娜,付玉荣,伊正君.结核分枝杆菌感染致脂代谢异常                          role of pyruvate kinase in carbon co-catabolism of Myco-
             及其机制研究进展[J].中华结核和呼吸杂志,2016,39                       bacterium tuberculosis[J]. J Biol Chem,2016,219(13):
             (7):548-550.                                        7060-7069.
        [11]  孙勇,李传友,许绍发.结核分枝杆菌耐药机制的研究进                     [25]  刘翠莲.miR-1 在吞噬活性调控以及肿瘤生长和转移中
             展[J].国际呼吸杂志,2010,30(24):295-301.                    的作用[D].杭州:浙江大学,2016.
        [12]  VILCHÈZE C,MOLLE V,CARRÈRE-KREMER S,et al.    [26]  VON BOTH U,BERK M,AGAPOW PM,et al. Mycobac-
             Phosphorylation of KasB regulates virulence and acid-fast-  terium tuberculosis exploits a molecular off switch of the
             ness in Mycobacterium tuberculosis[J]. PLoS Pathog,  immune system for intracellular survival[J]. Sci Rep,
             2014. DOI:10.137/journal.ppat.1004115.              2018,8(1):661.
        [13]  THOMPSON AP,STERNICKI L M,WEGENER KL,et al.   [27]  PIRES D,BERNARD EM,POMBO JP,et al. Mycobacte-
             生物素-抗结核药物新研究方向[J].江苏预防医学,2016,                      rium tuberculosis modulates miR-106b-5p to control ca-
             27(3):257-261.                                      thepsin s expression resulting in higher pathogen survival
        [14]  CASABON I,ZHU SH,OTANI H,et al. Regulation of      and poor T-cell activation[J]. Front Immunol,2017. DOI:
             the KstR2 regulon of Mycobacterium tuberculosis by a  10.3389/fimmu.2017.01819.
             cholesterol catabolite[J]. Mol Microbiol,2013,89(6):  [28]  ETNA MP,SINIGAGLIA A,GRASSI A,et al. Mycobac-
             1201-1212.                                          terium tuberculosis-induced miR-155 subverts autophagy
        [15]  SLAVIK MC,HOUSEWEART C,CRAMER C,et al. An-         by targeting ATG3 in human dendritic cells[J]. PLoS Pat-
             ticsus susceptibility profiles of Escherichia coli strains  hog,2018. DOI:10.1371/journal.ppat.1006790.
             lacking multidrug efflux plimp genes[J]. Antimicrob  [29]  彭丽娜,贾鹏.玛咖对力竭运动后老年模型大鼠肾脏线粒
             Agents Chemother,2011,45(4):1126-1136.              体呼吸功能及抗衰老能力的影响[J].中国组织工程研
        [16]  PREEZ ID,LOOTS DT. Altered fatty acid metabolism   究,2016,20(18):2711-2716.
             due to rifampicin-resistance conferring mutations in the  [30]  BLACK PA,WARREN RM,LOUW GE,et al. Energy
             rpoB gene of Mycobacterium tuberculosis:mapping the  metabolism and drug efflux in Mycobacterium tuberculo-
             potential of pharmaco-metabolomics for global health and  sis[J]. Antimicrob Agents Chemother,2014,58(5):2491-
             personalized medicine[J]. Omics,2012,16(11):596-603.  2503.
        [17]  KOUL A,VRANCKX L,DENDOUGA N,et al. Diary-     [31]  BOWN L,SRIVASTAVA SK,PIERCEY BM,et al. Myco-
             lquinolines are bactericidal for dormant mycobacteria as a  bacterial membrane proteins QcrB and AtpE:roles in ener-
             result of disturbed ATP homeostasis[J]. J Biol Chem,  getics,antibiotic targets,and associated mechanisms of re-
             2008,283(37):25273-25280.                           sistance[J]. J Membr Biol,2018,251(1):105-117.
        [18]  EOH H,RHEE KY. Multifunctional essentiality of succi-  [32]  BEMEY M,BERNEY-MEYER L,WONG KW,et al. Es-
             nate metabolism in adaptation to hypoxia in Mycobacteri-  sential roles of methionine and sadenosylmethionine in
             um tuberculosis[J]. Proc Natl Acad Sci USA,2013,110  the autarkic lifestyle of Mycobacterium tuberculosis[J].
             (16):6554-6559.                                     Proc Natl Acad Sci USA,2015,112(32):10008-10013.
        [19]  LEE YV,WAHAB HA,CHOONG YS. Potential inhibi-  [33]  VILJOEN A,RICHARD M,NGUYEN PC,et al. Cyclipo-
             tors for isocitrate lyase of Mycobacterium tuberculosis  stins and cyclophostin analogs inhibit the antigen 85C


        ·564  ·  China Pharmacy 2019 Vol. 30 No. 4                                   中国药房    2019年第30卷第4期
   140   141   142   143   144   145   146   147   148   149   150